Author(s) Details:
Abdullah M Nasrat
Department of Surgery, Balghsoon Clinic, Jeddah, Saudi Arabia.
Salwa AM Nasrat
Department of Physical Therapy, Cardiac Surgery Academy, Cairo, Egypt.
Randa M Nasrat
Department of Internal Medicine, Helwan General Hospital, Helwan, Egypt.
Mohammad M Nasrat
Department of Internal Medicine, Helwan General Hospital, Helwan, Egypt.
Sana Y Babiker
Faculty of Medicine, Elrazi University, Sudan.
This section is a part of the chapter: Helicobacter pylori and Hyperuricemia: Revisiting Gout Diagnosis in Young Adults with Normal Renal Function
Gout is caused by monosodium urate crystal deposition in joints and tissues. Risk factors include male sex; obesity; hypertension; alcohol intake; diuretic use; a diet rich in meat and seafood; chronic kidney disease; and a diet heavy in fructose-rich food and beverages (Clebak et al., 2020, Pittman and Bross, 1999). Gout is a very old disease which existed for thousands of years with joint swelling, pain or tenderness; the first description of symptoms of gout was found in the Egyptian medical papyri dating to 3000 years BC (Aluaak and Aluaak, 2015; Nasrat et al., 2015). Hyperuricemia has long been established as the major etiologic factor in gout (Gliozzi et al., 2015). Gout can also cause foot pain and disability in between flares, perhaps even before the first inflammatory flare occurs. Gout also happens more frequently in patients with some rheumatological diseases, such as psoriatic arthritis or osteoarthritis (Sivera et al., 2022). It has recently become the most common presentation of arthritis in developed countries; however, studies indicate that treatment of gout is still unsatisfactory (Cal-Kocikowska et al., 2015).
How to Cite
Nasrat, A. M., Nasrat, S. A., Nasrat, R. M., Nasrat, M. M., & Babiker, S. Y. (2025). Helicobacter pylori and Hyperuricemia: Revisiting Gout Diagnosis in Young Adults with Normal Renal Function. Medical Science: Trends and Innovations Vol. 4, 1–8. https://doi.org/10.9734/bpi/msti/v4/3641